Meals, eating, and nutrition
This is the 2nd Discussion Forum dedicated to this subject, and is a continuation of the previous forum on this subject. It was started on October 24, 2006, after what we call a "blogquake" happened -- namely, the software underlying this blog became unstable and shook everybody up!
Notices to our new readers:
1) There is another Discussion Forum for swapping recipes: click here to go there.
2) I will insist that there shall be no promotion of herbs or other concoctions that alledgedly lower blood sugar: any such comments (for example, "I took vitamin Z and my blood sugar miraculously returned to normal") will be deleted without warning.
3) I would suggest that anyone who is new to diabetes should read the articles at new to diabetes at DiabetesMonitor.com.4) Also, the folks who are regular readers and writers about the new diabetes drug Byetta have developed all sorts of strange terms that you'll gradually come to know, including "lizard spit", "gillyspitkins", "lizard of Az", "yellow spit road", "little brother" and "big brother", "hug island", and lots more. I think I'll dedicate a new Discussion Forum soon to all this new lingo...
4) Also, you can still read many of the earlier comments, and search for information throughout those forums, by using your webbrowser's FIND or SEARCH feature while at the earlier forums, or by searching at the advanced search feature that is at the Diabetes Monitor.
Please review and follow our usual ground rules, at Ground Rules. (To share your thoughts, use the "Comments" link immediately below. Instructions for entering comments may be found at How to Enter Comments.)
Bill the diabetesdoc
info@diabetesmonitor.com
Diabetes.Blog.Com is a blogcompanion to our main website, the Diabetes Monitor, which you can find at http://www.diabetesmonitor.com/
got my first box of dreamfields! (at shop-rite)
yummy stuff, and didn't affect my b.s. any more than anything else would.
my son picked up the box the other day and said,here's some pasta, i'll have this. i said no you won't! you eat the ronzoni! that stuff is MINE!
lol. i just love these boards, i've gotten such useful info here..... (Comment this)
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(Comment this)
I have two questions:
1) Is anyone taking byetta eating healthy small meals and still loosing weight?
I have read many of the blog entries about people using byetta not eating or reducing there food intake to unhealthy levels and have become concerned. I understand that byetta has appetite suppressant qualities which seem to lead help some loose weight. Many entries talk about people eating only a piece of fruit or a yogurt for a meal. I am concerned that this is not a healthy lifestyle. The body needs sufficient nutrients to thrive. I admit that I need to loose weight, but I do not want to rob my body of nutrients it needs to thrive by starving my body of nutrients. I also admit I eat fewer carbs than the nutritionist would like.
2) How long does byetta last in the system at a dose of 5 mg and or 10 mg?
I have lots of digestion issues and so my endo and I decided for me to start 5mg byetta before dinner only. That was 9-11-06 and so far I have not had nautia probably due to acid reflux rx. But I have noticed that while my bg reading 2 hrs after dinner is ok, but by 1 to 3 hours later it is going up, up, up. Example: yesterday blood glucose 169 before dinner, ate small healthy dinner, 120 2hrs after dinner, 140 3 hrs after dinner, and finally 188 4 hours after dinner (bedtime). Is this normal????? I thought the byetta was supposed to last about 6 hours.
Please let me know what you think. I would like to be well informed for my next meeting with my endo. I see him on Nov 20th and will be discussing how to best increase the byetta (add the morning injection) and still keep my digestive issues in control.
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so, here's the thing....if you are eating "small, healthy meals" you shoud be losing weight, regardless of the byetta. perhaps your endo has a diabetes educator or nutritionist who can review what and when you're eating to see what can be changed, and what shouldn't. so that if the byetta causes appetite suppression for you, you'll know what to cut out, and what to leave in.
you might look into the comments here (using the search feature) about acid/alkaline diet. i have foudn it has helped with my indigestion and reflux issues.
you would be surprised what foods cause an acid dsystem ,and by some that are alkaline. (Comment this)
I have not lost(or gained) even one lb since I began using byetta 1 month ago. I will be satisfied that at least my blood sugars do go down for at least a couple of hours each day. I will be discussing carefully and gradually increasing the byetta with my doctor(endo).
Thanks again for your input. (Comment this)
Thanks for the extra information about your diet. Also, thanks for telling us about the extra advantages of ACE inhibitors over beta blockers for diabetics. My comments about proteins not always being necessary were just meant to reassure a new spitkin about not feeling guilty about not eating protein(or "meat" as she said) for every breakfast. I only wanted to know who had given her that info, and did not mean to sound critical. That said, I also usually have some protein and fat each breakfast, usually dairy products. However, my husband makes wonderful waffles on weekends which we have with berries and whipped cream(a little fat there) and my bg readings two hours after are the same as after any other breakfast. The miracle of the spit! Also I am so happy to be able to eat one or two slices of pizza or 1/2 a baked potato whenever I want instead of just a few times a year as was the case before Byetta. Keep up the good work with your thoughtful answers to the new spitkins. (Comment this)
Can you send your husband this way? Sounds like a wonderful breakfast. I was not offended in any way at all but I sometimes say things in not the best way. Maybe I'm too blunt. I've been having a hard time of it lately and need to keep my mind open to new ideas and I'm no expert in the complex world of diabetes. Just a person who is searching for the answer to her own success and well being.
Hope you enjoyed your waffles this weekend, oh and in my mind the whipped cream(fat) should help to slow down the carb spike. How's that for a guiltfree attitude? Have a good day. (Comment this)
i'm not suffering any nausea, so have very little weight loss.
got the book ,alkalize or die. some interesting stuff in there. some surprising stuff, too. like lemon juice not being an acid causing product....
since making sure i eat more alkaline and less acid foods, my ulcer/acid reflux is much better. so that's one more thing i have to be grateful for, i guess. (Comment this)
i'm not suffering any nausea, so have very little weight loss.
got the book ,alkalize or die. some interesting stuff in there. some surprising stuff, too. like lemon juice not being an acid causing product....
since making sure i eat more alkaline and less acid foods, my ulcer/acid reflux is much better. so that's one more thing i have to be grateful for, i guess. (Comment this)
WASHINGTON (Reuters) -- Soy yogurt, especially with fruit in it, may help control both type 2 diabetes and high blood pressure, U.S. researchers report.
http://cnnstudentnews.cnn.com/2006/HEALTH/11/10/soy.yogurt.reut/index.html (Comment this)
http://cnnstudentnews.cnn.com/2006/HEALTH/11/10/soy.yogurt.reut/index.html (Comment this)
In principle I follow a similar diet plan, except I have not yet emphasized "Organic" in my meals. I have been vegetarian all my life. While staying on such a plan my A1c ranged from 7.8--7.2 (before May 2004 -- and then the qunituple bypass in May 2004), 6.0--6.5 (in late 2004), and 5.5--6.0 (in 2005 with lots of effort) without taking any meds. So the devil is in the details. To make it work requires a lot of fine turing and active vigilane -- details that are hard to discuss on a blog.
(Comment this)
I have been taking Byetta(5 pen)for the last week. So far I've been doing well. I find it very difficult to eat very much at all, which means Byetta is doing GREAT for appetite suppression. Today I took my injection ate a small breakfast (piece of whole grain toast, with about 1 tbs. of peanut butter on it). I believe it was not enough because my glucose tested at 73 by the time I got to work this morning. With no appetite I find it hard to force myself to eat. If I eat too much I tend to get horrible nausea. Any suggestions are welcome! (Comment this)
When I was young Linus Pauling made bold statements that Vitamin C was great because of its antioxidant properties. What was and antioxidant. Then I read a story in a Rand Think tank book. The story, paraphrased from memory went something like this.
In a lab in Alamogordo, New Mexico, on or about 1942, a nuclear scientist, name undisclosed, was conducting an experiment. On the other side of very thick lead glass shield was a table where two approximately 8 pound Plutonium hemispheres sat separated by several feet. This scientist is holding two sticks that allow him to push the two halves closer together. He does this.
Then he gets them to close. They begin to spark and produce obvious spark like between the two halves. You and I know now that neutrons were passing back and forth as a critical mass was fast approaching. He as a nuclear scientist knew this more keenly than we do.
He runs around the lead glass and places his hands between the two halves of Plutonium. The neutrons passed though his hands even more than his entire body. He received a dose of radiation far greater than 1,200 rads. This is fatal.
Other scientists asked the Doctors to explain what happened. The Doctors explained his death as oxidation. At the molecular level, a neutron impacted O2 molecules in his body breaking it into two molecules O –2 and O-2. This single atom of Oxygen is one of the most oxygen or oxidizing agents in chemistry. In fact the name oxidizing comes from Oxygen. This type of oxidizing burned this individual from the inside out a radiation burns. The single atom molecules of common oxidizers are free radicals.
Now I had a vision of an antioxidant like Vitamin C. In the presence of free radicals Vitamin C is typically oxidized instead of your internal body parts. Then the oxidized Vitamin C is flushed from the kidneys.
Over a lifetime some theorize that the radiation destruction is significant and that additional antioxidants are beneficial in reducing this destruction.
The story did not stop there, the military handlers asked the question of the Doctors. Is there anything we can do to keep our soldiers from being killed after radiation exposure? 50% of monkeys exposed to 600 rads died. Monkeys given Cysteine and amino acid antioxident could withstand 1,200 rads before 50% of the monkeys died. N-acetyl-L-cysteine is also a great antioxident.
N-acetyl-L-cysteine is used to help minimize the damage caused to the liver from an overdose of acetaminophen. The liver plays a big part in some of the metobolic disorders of a t2 diabetic.
Frank Roy, is blogging on the Januvia side reported that he supplemented N-acetyl-L-cysteine, or NAC. I have come to respect his research and logic. So Frank, why do you take this suplemen? Do you have some receint t2 research linking NAC to diabetes health?
I, John Meek, suplement 500-1,000 Vitimin C, and 1,000 Units E for their antioxicent properties. Once however, I thought that we could improve health by a amino acid antioxident, water soluable antioxidnent, and a fat soluable antioxicent. By the way I don’t recommend 1,000 Units of E. 400 Units is more reasonable.
That is why I believe in antioxidents and free radical scavengers.
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I'm not sure what type of bagel you ate but if you want to continue eating a bagel like the one you ate yesterday try eating only 1/2 of the bagel with cream cheese. Then add a fresh fruit to supplement. I also don't know what type of peanut butter cracker you ate but if you ate a whole grain cracker with peanut butter added and added some veggies you might have better results. Hope this helps. (Comment this)
JM "So Frank, why do you take this suplement? Do you have some receint t2 research linking NAC to diabetes health?"
It like this type 2 DMs generally have a lot of what is termed oxidative stress. Oxidative Stress and Stress-Activated Signaling Pathways: A Unifying Hypothesis of Type 2 Diabetes - http://edrv.endojournals.org/cgi/content/full/23/5/599
"In both type 1 and type 2 diabetes, the late diabetic complications in nerve, vascular endothelium, and kidney arise from chronic elevations of glucose and possibly other metabolites including free fatty acids (FFA). Recent evidence suggests that common stress-activated signaling pathways such as nuclear factor-{kappa}B, p38 MAPK, and NH2-terminal Jun kinases/stress-activated protein kinases underlie the development of these late diabetic complications. In addition, in type 2 diabetes, there is evidence that the activation of these same stress pathways by glucose and possibly FFA leads to both insulin resistance and impaired insulin secretion. Thus, we propose a unifying hypothesis whereby hyperglycemia and FFA-induced activation of the nuclear factor-{kappa}B, p38 MAPK, and NH2-terminal Jun kinases/stress-activated protein kinases stress pathways, along with the activation of the advanced glycosylation end-products/receptor for advanced glycosylation end-products, protein kinase C, and sorbitol stress pathways, plays a key role in causing late complications in type 1 and type 2 diabetes, along with insulin resistance and impaired insulin secretion in type 2 diabetes. Studies with antioxidants such as vitamin E, {alpha}-lipoic acid, and N-acetylcysteine suggest that new strategies may become available to treat these conditions. ... he molecular mechanisms whereby oxidative stress causes diabetic complications are undefined. In a variety of tissues, hyperglycemia and elevated FFA result in the generation of ROS and RNS, leading to increased oxidative stress. In the absence of an appropriate compensatory response from the endogenous antioxidant network, the system becomes overwhelmed (redox imbalance), leading to the activation of stress-sensitive signaling pathways, such as NF-{kappa}B, p38 MAPK, JNK/SAPK, PKC, AGE/RAGE, sorbitol, and others. The consequence is the production of gene products, such as VEGF and others, which cause cellular damage and are ultimately responsible for the long-term complications of diabetes. In addition, activation of the same or similar pathways appears to mediate insulin resistance and impaired insulin secretion. It is our view that there appears to be a common biochemical basis that involves oxidative-stress-induced activation of stress-sensitive signaling pathways. Thus, the use of antioxidants may be very important in preventing activation of these pathways." This article has been cited in 207 subsequent articles. Links to the citing articles are in the second one listed in the following: scholar.google.com search for the terms: "oxidative stress"+diabetes+ "endogenous antioxidant"+synthesis+glutathione+ superoxide+dismutase which resulted in 367 finds - http://tinyurl.com/yesh9f. While I only take a minimum of vitamin C that is in a multiple vitamin/mineral, I do take other supplements like NAC which help to produce natural/endogenous antioxidants. Selenium is one of the components of the recipe if you will. I try to take a global approach to address the various aspects of the metabolic syndrome and antioxidants are a part of it. That's it in a nut shell.
Frank (Comment this)
Eating complex carbs instead of simple carbs makes a big difference for me. (Comment this)
**sigh** yes, you're right, i know. i have no idea what i ate wrong (if anything) but i'll keep track and figure it out.
and
to frank roy: that's a mighty big nutshell. (Comment this)
Tami said to frank roy: that's a mighty big nutshell.
Agreed! ;) But maybe John Meek can break it down.
I was expecting him to give some comment(s).
Frank (Comment this)
Thanks for your confidence Tami, and thanks for the specific informaiton Frank. It is a pleasure to find someone looking at t2 diabetes from a nutrition as well as drungs.
This discussion is about anti-oxidents and why we believe that they are good for your body in general and Frank has identified items he specifically thinks that they benefit in relationship to diabetes.
The extent an indiviudual suplements anitoxidents is an informed personal choice that should involve research and some communicaiton with your doctor.
Frank and I have decided that it is beneficial and use different sourses to acomplish it. I will introduce other specific topics in nutrition later. For example, some over the counter things can be used to combat insulin resistance.
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Our modern patent laws allow for great drugs to be tested and used, while non-patentable drugs to be untested and ignored. In the realm of non-patentable nutrients and natural substances are real pharmacological effects. As all good things this started with a story. I was watching a drug trial from Insmed where they were testing IGF-1 as an insulin replacement for diabetics. Deep in the drug information package there was a new compound that they added to the preparation, d-Chiro-Inositol. In there literature they stated that d-Chiro-Inositol was added because it increased insulin efficiency. In the early research it was stated that Type 1 diabetics could use 80% of the insulin with d-Chiro-Inositol to be equal to 100% of the insulin alone. This is not the time to talk about IGF-1, Insulin like Growth Hormone Factor 1, but the insulin sensitizer d-Chiro-Inositol.
D-Chiro-Inositol is found in nature in several foods, lecithin and carob. It is a commercial emulsifier. It is not a drug; it is a food and a food additive. A Chinese company will sell it to you. It is relatively expensive per kilogram. I could not figure out how to get the money to them and get it shipped, so I have never purchased the pure form. There is no market for the drug companies to provide this. For some reason the health food stores have not picked up on it.
US patent number 6492339 provides compositions comprising synergistically effective amounts of D-Chiro-Inositol and sulfonylureas. US patent number 5427956 provides a test for insulin resistance by: assaying the urine for the presence of D-Chiro-Inositol,
And classifying the individual, as insulin-resistant if the level of D-Chiro-Inositol in the urine sample is equal to or less than a predetermined level of urinary D-Chiro-Inositol, which is indicative of insulin resistance. D-Chiro-Inositol is found in type 2 diabetics in excess amounts in the urine, so a safe theory extrapolation is that the high amounts of d-Chiro-Inositol in the urine is a chemical byproduct of insulin resistance. The two instances of drug inclusion seem to support that supplementing this inositol can increase insulin efficiency. If you look into it you will find that it is beneficial and that there is no risk in supplementation. It is basically considered a food or food additive.
Since I could not find obtain the isomer I started looking at foods that had higher concentrations of it. You have Inositol, which has a lot of inositol isomers inclusive of d-Chiro-Inositol, lecithin has 2-6% of d- Chiro-Inositol, and US patent number 6699511 from a pinitol-containing carob extract, which looks to be 10 to 11% pinitol. Pinitol is (3-O-methyl-D-chiro-inositol, which behaves similarly to D-Chiro-Inositol.
US patent number 5763392 discusses a method for lowering the plasma glucose levels of diabetics by administering myo-inositol. Myo-inositol shares similarities to D-Chiro-Inositol.
This is an unscientific un-proven personal theory by John Meek of D-Chiro-Inositol and insulin resistance. In the metabolic syndrome individual the blood is thickened by LDL cholesterol and glucose. The blood becomes more like syrup and less like water. A study of LDL cholesterol reveals that glucose actually forms a bond to the LDL cholesterol. Insulin resistance of this context happens when the insulin molecule rests on the insulin receptor and sets up a chemical reaction that through another pathway sends a chemical catcher to the wall of the cell and attempts to grab a glucose molecule and bringing it into the cell. This is a normal person.
Examine the molecules of D-Chiro-Inositol and glucose in a molecular model and you will se that they are mirror images of roughly the same signatures. They fit together like a glove. I postulate that the D-Chiro-Inositol is the catcher’s mitt of the cell for the glucose. In the metabolic syndrome the glucose gets in a tug of war with the LDL cholesterol electrochemical bond and thickened blood. This blood becomes more like syrup. The thickness of the blood, viscosity, and blood pressure cause the D-Chiro-Inositol component in this cell to be pulled out of the cell and not pulling in the glucose into the cell. Therefore, more and more D-Chiro-Inositol is excreted. At some point a lot of the cells are depleted and a critical component of the electro chemical signaling for the insulin receptor is gone. Your have insulin resistance.
Supplementing D-Chiro-Inositol will not beat your insulin resistance, but you might get 20% efficiently change by supplementing it. It is just one of many little things I do to improve insulin resistance. Since this is not a drug the drug companies are not going to study it separately. That’s why we need College funding to study non-drug medical treatments. If you attempt to eat inositol or lecithin to get this small percentage isomer, then you will need to eat a lot of it. I have not idea what amount you should take but 2-6% is a small occurrence, so we are talking about 12 to 25 grams or teaspoon to a tablespoon. I get raw graduals of Lecithin from the health food store and on occasion have purchased powered inositol. The good news is there is no data on toxicity, this is a food. The bad news is how do you get standardized controllable amounts in this way. Pinitol, myo-inositol and D-Chiro-Inositol have similar properties and should work similarly. If D-Chiro-Inositol is in the blood and excreted we run the risk that increasing D-Chiro-Inositol in the intestines increases the blood level and is excreted. So have the risk that you can spend your money and get not benefit from this theory. But if that were true then the two drugs would not have included it in their concoctions.
Later we need to talk about blood thinners, risks, and benefits.
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Most of the research articles on d-chiro-inositol that I found related to polycystic ovary syndrome (PCOS). There are some excerpts toward the end on T2 DM and is good to know information.
Ovulatory and Metabolic Effects of d-Chiro-Inositol in the Polycystic Ovary Syndrome -
http://content.nejm.org/cgi/content/abstract/340/17/1314
The original article was published in 1999, but there are some more recent citations of it like a few publisehed in 2006.
D-Chiro-Inositol and Insulin Action -
http://tinyurl.com/u83jo
"Almost all of the inositol in nature is myo-inositol. However, several preparations of phosphoglycan molecules that appear to mediate insulin action as second messengers contain D-chiro-inositol ... . This laboratory has shown that diabetic subjects excrete 5-40 times as much D-chiro-inositol in the urine as normal subjects and that the rate of excretion depends critically on the degree of diabetic control. Plasma D-chiro-inositol levels are influenced by insulin treatment and are a strong predictor of hypertriglyceridemia, the most common lipid abnormality in diabetic patients. D-chiro-inositol can be obtained from the diet in the form of pinitol, a methyl inositol found in legumes. Feeding diabetic patients D-chiro-inositol from soy results in reductions in plasma insulin levels. The effects of D-chiro-inositol on diabetic subjects is being studied."
D-chiro-inositol metabolism in diabetes mellitus - http://tinyurl.com/v7xom. "Median urinary D-chiro-inositol excretion, which was 2.1 mumol/day in nondiabetics, was substantially increased to 12 mumol/day in non-insulin
dependent diabetes and to 74 mumol/day in insulin-dependent diabetes. Urinary D-chiro-inositol was strongly correlated with fasting plasma glucose, glycated hemoglobin, and urinary glucose. The renal clearance of D-chiro-inositol was selectively elevated in both non-insulin-dependent and insulin-dependent diabetes when compared with the clearances of L-chiro-inositol or myo-inositol and exceeded the glomerular filtration rate in 71% of the diabetics but in none of the nondiabetics. In poorly controlled diabetic patients insulin treatment reduced urinary D-chiro-inositol losses by 63% and increased plasma levels by 8.8-fold. The metabolism of D-chiro-inositol is abnormal in diabetes and appears to be influenced by short- and long-term metabolic control."
Frank (Comment this)
HMG coA reductase, an enzyme inhibitor, has been isolated from the Lobster mushroom. HMG coA reductase inhibitors have been implicated in reducing plaque in large to medium sized arteries and thus reducing death from coronary disease. Hypomyces lactifluorum produces skyrin, which may prove useful in treating diabetic hyperglycemia.
“In this regard, development of structural and functional glucagon receptor antagonists represents a potential approach to decrease hepatic glucose production and lower blood glucose in patients with diabetes. The fungal bisanthroquinone skyrin, isolated from Talaromyces wortmannin, inhibits glucagon-stimulated cAMP formation and glucose output from rat and human hepatocytes. Skyrin does not appear to interfere with glucagon receptor binding in CHO cells or hepatocytes. Whether skyrin will also reduce blood glucose in normal or diabetic rodents in vivo remains unclear. See Effects of skyrin, a receptor-selective glucagon antagonist, in rat and human hepatocytes. Diabetes. 2000 Dec;49(12):2079-86”
HMG-CoA reductase (or 3-hydroxy-3-methyl-glutaryl-CoA reductase or HMGR) is the first enzyme (EC 1.1.1.88) of the HMG-CoA reductase pathway, the metabolic pathway that produces cholesterol and various other biomolecules. Drugs which inhibit HMG-CoA reductase, known collectively as HMG-CoA reductase inhibitors (or "statins"), are used in to lower serum cholesterol as a means of reducing the risk for cardiovascular disease.
http://diabetes.diabetesjournals.org/cgi/reprint/49/12/2079.pdf “Effects of Skyrin, a Receptor-Selective Glucagon Antagonist, in Rat and Human Hepatocytes”
http://www.glucagon.com/glucagon_receptor_antagonists.htm “Glucagons Receptor Antagonists”
I’m not looking for much discussion on this. This is another interesting herb. I made myself a soup of 15 grams of Lobster Mushrooms in a can on beef broth and it seemed to help my blood sugar. I did not like the soup very much and that much would cost a fortune. I ground up the lobster mushrooms and put them into 00 capsules and estimated that I had approximately 500 mg of lobster mushroom. That little of the herb did not seem to make any difference. Lobster mushrooms are a fungus growing on another fungus. It is the secondary fungus that has the red cooler and includes the active ingredient, so most of the lobster mushroom is just a mushroom. A very small active ingredient Skyrin is present. This is another food with real pharmacological action.
The implication of being both a Glucagon antagonist and reducing cholesterol in the same chemical attacks two of the major issues of diabetic metabolic syndrome: cholesterol and lowering blood sugar. Implication of reducing plaque in large to medium sized arteries, if true, is a very good mix for a drug. Could this work synergistically with DPP-4 inhibitors?
(Comment this)
A million dollar word - Inositolphosphoglycan.
Some researchers see the connection between inositolphosphoglycan mediators and GLP-1.
This is part of the direction that past research on D-chiro-insoitol has currently taken.
Frank (Comment this)
Usual Evening meal: small portions, healthy cooking style, high fiber. Mix of protein, fat, and carb (usually about 35 gr from grain, and some from veg or fruit)
I find that after my evening meal my bg readings continue to go up after the 2 the usual hours. It peaks at 160-225 about 5 hours after the meal. This confuses me since my fasting bg is now about 118. The Lantus has helped with the fasting bg but has not had much effect on the ones after the evening meal. After breakfast and lunch same type of response but does not go up as high as at night.
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When they put me on Protonix for my acid reflux I discovered that it allowed me to override the appetite suppression of the byetta, I felt too darn good. I discontinued it after 3 weeks during which time I was eating alkaline producing foods and the acid reflux pretty much went away. If I should eat too many acid producing foods, which is easy to do, and I get acid reflux again I use papaya enzymes or chew mint gum.
So I guess what I'm asking you to think about is - are you eating more food because you are feeling better and not stopping when you no longer feel hungry? That would raise your numbers as it did for me. On page 6 of this island, in previous, is a list of alkaline producing foods which might help you too. I'm also eating more lower glycemic index foods, when I do my numbers are great at bed time.
One more thing, the best time to test after eating a spit meal is 3-4 hrs after because of the slower gastric emptying caused by the spit. Hope this helps. (Comment this)
I thought I would let you all know... it helps me keep my cravings for fried foods under control. Or at least it makes them more healthy. If interested here's the website of the brand that my family and I have. http://www.nesco.com/products/?category=600 (Comment this)
No I am not eating more food and I do watch the foods I eat and avoid acid reflux trigger foods. I have had severe acid reflux for 10 years. It is so bad that even water first thing in the morning before any food will come right back up. I am not wanting to stop taking the protonix.
I mostly am trying to figure out why my after meal readings continue to go up so long after eating a small healthy meal and if protonix is delaying the food digestion should I be taking my after meal glucose reading at 3 or 4 hours instead of 2 hours.
clk (Comment this)
I'm sorry, I'm in such a spit mode I automatically think that way, but it looks like you are not on the spit. I can't say about Protonix delaying anything but I do know that I've have great success with eating alkaline producing foods. The name of the book I read to learn about alkaline eating is called "Alkalize or Die" by Theodore Broody. Some natural food stores carry it in their libraries or you can get it from Amazon. Good Luck. (Comment this)
Geri, Would a barley, black bean and chicken dish (with vegies thrown in) qualify as a complex fiber? I have a wonderful salad recipe that I would be willing to share on the recipe page. Bonnie (Comment this)
I've tried several receipes and they have all been good but my numbers didn't do so great with a few of them. But we are all different. I'm looking forward to giving it a try. (Comment this)
So before breakfast, my bg was 139, 2 hours after with a carb load of only 14 grams it was 217...
The information GP (and I have seen elsewhere) gave me was that is shouldn't be more than 50 different two hours after a meal thatn t was before (and should be <180); and that the less carbs you eat the lower the delta should be...am I confused or have misread something?
Does the delta of 50 only work if you are non-diabetic?
(Comment this)
It's not just the carb count that is important but the type of carb that you eat that is probably more important. White or processed carbs spike high and fast whereas whole grain,not wheat but whole grain, carbs with lots of fiber are slower to rise and give us a more even level. Processed carbs take all the fiber out. I try to eat low glycemic index carbs which help keep the bg levels more level. Hope this helps a little. (Comment this)
The carb was a piece of no-soy 12 grain bread with my scrambled eggs (no milk added) (Comment this)
What are the total carbs and the fiber content? If it doesn't have at least 4 grams of fiber per serving it may not be the best bread for you. You may have to do some experimentation to see what works best for you since we are all different. Make sure it is whole grain. 12 grain may still not be whole grain. It's alot of trying this and that to see what works for you.
In the beginning we all struggle with trying to understand what we are and are not suppose to do. (Comment this)
You mentioned that you are an advocate for a "low G.I" diet. How many Calories, carbs, proteins and fats is that on an average day? (Comment this)
It has enough fiber, but who knows..I'll pair it with something else that hasn't shown a spike and see if the same thing happens..I am thinking it is a dan effect that happens later in the day (10am ish) for me than for others? Does the bosy figure dawn by when you go to be??? how does that work (Comment this)
I didn't mention that because you were eating an egg, protein. Proteins and fats will slow down spikes. It could also be the fact that as you get moving in the am your body releases more glucose to meet the demands but if you are insulin resistent it doesn't get used properly and it hangs around. All kinds of reasons so sometimes it's hard to pinpoint just one cause. Do you exercise? (Comment this)
I do exercise, Gym M-F in the pool (unless its under 20 degrees, then we don't go to the gym-not enough time to defrost the car both ways and damp hair at 20 is asking for trouble)
I did not expect the bg to be so unpredictable...
(Comment this)